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Katsuyoshi Sunaga

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Chemicals & Drugs

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Katsumi Tsuchiya; Hisao Tajima; Toyoyasu Kuwae; Takao Takeshima; Toshiya Nakano; Masaharu Tanaka; Katsuyoshi Sunaga; Yoko Fukuhara; Kenji Nakashima; Eisaku Ohama; Hideki Mochizuki; Yoshikuni Mizuno; Nobuo Katsube; Ryoichi Ishitani
Pro-apoptotic protein glyceraldehyde-3-phosphate dehydrogenase promotes the formation of Lewy body-like inclusions.
Hisao Tajima; Katsuyoshi Sunaga; Masaharu Tanaka; Toyoyasu Kuwae; Nobuo Katsube
Overexpression of glyceraldehyde-3-phospahe dehydrogenase is not involved in 5-hydroxytryptamine (5-HT)-induced necrosis in cultured cerebrocortical neurons.
Katsumi Tsuchiya; Hisao Tajima; Mitsunori Yamada; Hitoshi Takahashi; Toyoyasu Kuwae; Katsuyoshi Sunaga; Nobuo Katsube; Ryoichi Ishitani
Disclosure of a pro-apoptotic glyceraldehyde-3-phosphate dehydrogenase promoter: anti-dementia drugs depress its activation in apoptosis.

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All Publications

2005: Katsumi Tsuchiya; Hisao Tajima; Toyoyasu Kuwae; Takao Takeshima; Toshiya Nakano; Masaharu Tanaka; Katsuyoshi Sunaga; Yoko Fukuhara; Kenji Nakashima; Eisaku Ohama; Hideki Mochizuki; Yoshikuni Mizuno; Nobuo Katsube; Ryoichi Ishitani
Pro-apoptotic protein glyceraldehyde-3-phosphate dehydrogenase promotes the formation of Lewy body-like inclusions.
The European journal of neuroscience 2005;21(2):317-26.
2004: Hisao Tajima; Katsuyoshi Sunaga; Masaharu Tanaka; Toyoyasu Kuwae; Nobuo Katsube
Overexpression of glyceraldehyde-3-phospahe dehydrogenase is not involved in 5-hydroxytryptamine (5-HT)-induced necrosis in cultured cerebrocortical neurons.
Biological & pharmaceutical bulletin 2004;27(8):1224-7.
2004: Katsumi Tsuchiya; Hisao Tajima; Mitsunori Yamada; Hitoshi Takahashi; Toyoyasu Kuwae; Katsuyoshi Sunaga; Nobuo Katsube; Ryoichi Ishitani
Disclosure of a pro-apoptotic glyceraldehyde-3-phosphate dehydrogenase promoter: anti-dementia drugs depress its activation in apoptosis.
Life sciences 2004;74(26):3245-58.
1999: Nobuo Katsube; Katsuyoshi Sunaga; H Aishita; De-Maw Chuang; Ryoichi Ishitani
ONO-1603, a potential antidementia drug, delays age-induced apoptosis and suppresses overexpression of glyceraldehyde-3-phosphate dehydrogenase in cultured central nervous system neurons.
The Journal of pharmacology and experimental therapeutics 1999;288(1):6-13.
1998: Ryoichi Ishitani; Masaharu Tanaka; Katsuyoshi Sunaga; Nobuo Katsube; De-Maw Chuang
Nuclear localization of overexpressed glyceraldehyde-3-phosphate dehydrogenase in cultured cerebellar neurons undergoing apoptosis.
Molecular pharmacology 1998;53(4):701-7.
1997: Ryoichi Ishitani; Katsuyoshi Sunaga; Masaharu Tanaka; H Aishita; De-Maw Chuang
Overexpression of glyceraldehyde-3-phosphate dehydrogenase is involved in low K+-induced apoptosis but not necrosis of cultured cerebellar granule cells.
Molecular pharmacology 1997;51(4):542-50.
1996: Nobuo Katsube; Katsuyoshi Sunaga; De-Maw Chuang; Ryoichi Ishitani
ONO-1603, a potential antidementia drug, shows neuroprotective effects and increases m3-muscarinic receptor mRNA levels in differentiating rat cerebellar granule neurons.
Neuroscience letters 1996;214(2-3):151-4.
1996: Ryoichi Ishitani; M Kimura; Katsuyoshi Sunaga; Nobuo Katsube; Masaharu Tanaka; De-Maw Chuang
An antisense oligodeoxynucleotide to glyceraldehyde-3-phosphate dehydrogenase blocks age-induced apoptosis of mature cerebrocortical neurons in culture.
The Journal of pharmacology and experimental therapeutics 1996;278(1):447-54.
1996: Ryoichi Ishitani; Katsuyoshi Sunaga; A Hirano; P Saunders; Nobuo Katsube; De-Maw Chuang
Evidence that glyceraldehyde-3-phosphate dehydrogenase is involved in age-induced apoptosis in mature cerebellar neurons in culture.
Journal of neurochemistry 1996;66(3):928-35.
1995: Katsuyoshi Sunaga; H Takahashi; De-Maw Chuang; Ryoichi Ishitani
Glyceraldehyde-3-phosphate dehydrogenase is over-expressed during apoptotic death of neuronal cultures and is recognized by a monoclonal antibody against amyloid plaques from Alzheimer's brain.
Neuroscience letters 1995;200(2):133-6.
1993: Katsuyoshi Sunaga; De-Maw Chuang; Ryoichi Ishitani
Tetrahydroaminoacridine increases m3-, but not m2-, muscarinic acetylcholine receptor mRNA levels in differentiating cerebellar granule cells.
Neuroscience letters 1993;163(1):27-30.
1993: Katsuyoshi Sunaga; De-Maw Chuang; Ryoichi Ishitani
Autoradiographic demonstration of an increase in muscarinic cholinergic receptors in cerebellar granule cells treated with tetrahydroaminoacridine.
Neuroscience letters 1993;151(1):45-7.
1993: Katsuyoshi Sunaga; De-Maw Chuang; Ryoichi Ishitani
Tetrahydroaminoacridine is neurotrophic and promotes the expression of muscarinic receptor-coupled phosphoinositide turnover in differentiating cerebellar granule cells.
The Journal of pharmacology and experimental therapeutics 1993;264(1):463-8.