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Amir Zalcenstein

Research Profile

Chemicals & Drugs

Physiology

Anatomy

Cell Line

Disorders

DNA Damage

Living Beings

Mouse

Procedures

Chromatin Immunoprecipitation

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Publications

TOP 3
Amir Zalcenstein; Lilach Weisz; Perry Stambolsky; Jair Bar; Varda Rotter; Moshe Oren
Repression of the MSP/MST-1 gene contributes to the antiapoptotic gain of function of mutant p53.
J Bar; N Lukaschuk; Amir Zalcenstein; S Wilder; R Seger; Moshe Oren
The PI3K inhibitor LY294002 prevents p53 induction by DNA damage and attenuates chemotherapy-induced apoptosis.
Lilach Weisz; Amir Zalcenstein; Perry Stambolsky; Yehudit Cohen; Naomi Goldfinger; Moshe Oren; Varda Rotter
Transactivation of the EGR1 gene contributes to mutant p53 gain of function.

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All Publications

2006: Amir Zalcenstein; Lilach Weisz; Perry Stambolsky; Jair Bar; Varda Rotter; Moshe Oren
Repression of the MSP/MST-1 gene contributes to the antiapoptotic gain of function of mutant p53.
Oncogene 2006;25(3):359-69.
2005: J Bar; N Lukaschuk; Amir Zalcenstein; S Wilder; R Seger; Moshe Oren
The PI3K inhibitor LY294002 prevents p53 induction by DNA damage and attenuates chemotherapy-induced apoptosis.
Cell death and differentiation 2005;12(12):1578-87.
2004: Lilach Weisz; Amir Zalcenstein; Perry Stambolsky; Yehudit Cohen; Naomi Goldfinger; Moshe Oren; Varda Rotter
Transactivation of the EGR1 gene contributes to mutant p53 gain of function.
Cancer research 2004;64(22):8318-27.
2003: Amir Zalcenstein; Perry Stambolsky; Lilach Weisz; Martina Müller; David Wallach; Tanya M Goncharov; Peter H Krammer; Varda Rotter; Moshe Oren
Mutant p53 gain of function: repression of CD95(Fas/APO-1) gene expression by tumor-associated p53 mutants.
Oncogene 2003;22(36):5667-76.
2003: Xinjiang Wang; Amir Zalcenstein; Moshe Oren
Nitric oxide promotes p53 nuclear retention and sensitizes neuroblastoma cells to apoptosis by ionizing radiation.
Cell death and differentiation 2003;10(4):468-76.
1999: A Elkeles; T Juven-Gershon; David Israeli; S Wilder; Amir Zalcenstein; Moshe Oren
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
Molecular and cellular biology 1999;19(4):2594-600.

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