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GE DeLander

Research Profile

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Publications

TOP 3
GE DeLander
Rules of the road.
GE DeLander; E Schött; Ernst Brodin; Bertil B. Fredholm
Spinal expression of mRNA for immediate early genes in a model of chronic pain.
GE DeLander; E Schött; Ernst Brodin; Bertil B. Fredholm
Temporal changes in spinal cord expression of mRNA for substance P, dynorphin and enkephalin in a model of chronic pain.

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All Publications

2012: GE DeLander
Rules of the road.
American journal of pharmaceutical education 2012;76(5):76.
1997: GE DeLander; E Schött; Ernst Brodin; Bertil B. Fredholm
Spinal expression of mRNA for immediate early genes in a model of chronic pain.
Acta physiologica Scandinavica 1997;161(4):517-25.
1997: GE DeLander; E Schött; Ernst Brodin; Bertil B. Fredholm
Temporal changes in spinal cord expression of mRNA for substance P, dynorphin and enkephalin in a model of chronic pain.
Acta physiologica Scandinavica 1997;161(4):509-16.
1997: H Choi; Thomas F Murray; GE DeLander; W K Schmidt; Jane V Aldrich
Synthesis and opioid activity of [D-Pro10]dynorphin A-(1-11) analogues with N-terminal alkyl substitution.
Journal of medicinal chemistry 1997;40(17):2733-9.
1997: Seksiri Arttamangkul; Jane E Ishmael; Thomas F Murray; David K Grandy; GE DeLander; B L Kieffer; Jane V Aldrich
Synthesis and opioid activity of conformationally constrained dynorphin A analogues. 2. Conformational constraint in the "address" sequence.
Journal of medicinal chemistry 1997;40(8):1211-8.
1996: Gary Keil; GE DeLander
Altered sensory behaviors in mice following manipulation of endogenous spinal adenosine neurotransmission.
European journal of pharmacology 1996;312(1):7-14.
1995: Gary Keil; GE DeLander
Time-dependent antinociceptive interactions between opioids and nucleoside transport inhibitors.
The Journal of pharmacology and experimental therapeutics 1995;274(3):1387-92.
1995: Seksiri Arttamangkul; Thomas F Murray; GE DeLander; Jane V Aldrich
Synthesis and opioid activity of conformationally constrained dynorphin A analogues. 1. Conformational constraint in the "message" sequence.
Journal of medicinal chemistry 1995;38(13):2410-7.
1994: Gary Keil; GE DeLander
Adenosine kinase and adenosine deaminase inhibition modulate spinal adenosine- and opioid agonist-induced antinociception in mice.
European journal of pharmacology 1994;271(1):37-46.
1993: K R Snyder; Thomas F Murray; GE DeLander; Jane V Aldrich
Synthesis and opioid activity of dynorphin A-(1-13)NH2 analogues containing cis- and trans-4-aminocyclohexanecarboxylic acid.
Journal of medicinal chemistry 1993;36(13):1921-2.
1993: K R Snyder; Thomas F Murray; GE DeLander; Jane V Aldrich
Synthesis and opioid activity of dynorphin A-(1-13)NH2 analogues containing cis- and trans-4-aminocyclohexanecarboxylic acid.
Journal of medicinal chemistry 1993;36(8):1100-3.
1992: GE DeLander; Henry I Mosberg; F Porreca
Involvement of adenosine in antinociception produced by spinal or supraspinal receptor-selective opioid agonists: dissociation from gastrointestinal effects in mice.
The Journal of pharmacology and experimental therapeutics 1992;263(3):1097-104.
1992: H Choi; Thomas F Murray; GE DeLander; V Caldwell; Jane V Aldrich
N-terminal alkylated derivatives of [D-Pro10]dynorphin A-(1-11) are highly selective for kappa-opioid receptors.
Journal of medicinal chemistry 1992;35(24):4638-9.
1992: K R Snyder; S C Story; M E Heidt; Thomas F Murray; GE DeLander; Jane V Aldrich
Effect of modification of the basic residues of dynorphin A-(1-13) amide on kappa opioid receptor selectivity and opioid activity.
Journal of medicinal chemistry 1992;35(23):4330-3.
1992: S C Story; Thomas F Murray; GE DeLander; Jane V Aldrich
Synthesis and opioid activity of 2-substituted dynorphin A-(1-13) amide analogues.
International journal of peptide and protein research 1992;40(2):89-96.
1992: Gary Keil; GE DeLander
Spinally-mediated antinociception is induced in mice by an adenosine kinase-, but not by an adenosine deaminase-, inhibitor.
Life sciences 1992;51(19):PL171-6.
1991: GE DeLander; J J Wahl
Descending systems activated by morphine (ICV) inhibit kainic acid (IT)-induced behavior.
Pharmacology, biochemistry, and behavior 1991;39(1):155-9.
1989: GE DeLander; J J Wahl
Morphine (intracerebroventricular) activates spinal systems to inhibit behavior induced by putative pain neurotransmitters.
The Journal of pharmacology and experimental therapeutics 1989;251(3):1090-5.
1989: GE DeLander; J J Wahl
In vivo characterization of phencyclidine/sigma agonist-mediated inhibition of nociception.
European journal of pharmacology 1989;159(2):149-56.
1988: GE DeLander; J J Wahl
Behavior induced by putative nociceptive neurotransmitters is inhibited by adenosine or adenosine analogs coadministered intrathecally.
The Journal of pharmacology and experimental therapeutics 1988;246(2):565-70.
1987: GE DeLander; C J Hopkins
Interdependence of spinal adenosinergic, serotonergic and noradrenergic systems mediating antinociception.
Neuropharmacology 1987;26(12):1791-4.
1987: GE DeLander; C J Hopkins
Involvement of A2 adenosine receptors in spinal mechanisms of antinociception.
European journal of pharmacology 1987;139(2):215-23.
1986: GE DeLander; C J Hopkins
Spinal adenosine modulates descending antinociceptive pathways stimulated by morphine.
The Journal of pharmacology and experimental therapeutics 1986;239(1):88-93.