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William Armstead
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8
Cines, Douglas
6
Higazi, Abd Al-Roof
5
Wilderman, Michael
4
Philip, Shaji
3
Baranov, Dimitry
2
Bdeir, Khalil
2
Chen, Xiao-Han
2
Smith, Douglas
2
Stein, Sherman
2
Vavilala, Monica
2
Kurth, Dean
2
ROSSBERG, MARK
1
Udomphorn, Yuthana
1
Zaitsev, Sergei
1
Zimmerman, Jerry
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All Publications
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2009: Philip Shaji; Chaiwat Onuma; Udomphorn Yuthana; Moore Anne; Zimmerman Jerry J; Armstead William; Vavilala Monica S
Variation in cerebral blood flow velocity with cerebral perfusion pressure >40 mm Hg in 42 children with severe traumatic brain injury.
Critical care medicine 2009;37(11):2973-8.
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2009: Armstead William M; Ganguly Kumkum; Kiessling John W; Chen Xiao-Han; Smith Douglas H; Higazi Abd A R; Cines Douglas B; Bdeir Khalil; Zaitsev Sergei; Muzykantov Vladimir R
Red blood cells-coupled tPA prevents impairment of cerebral vasodilatory responses and tissue injury in pediatric cerebral hypoxia/ischemia through inhibition of ERK MAPK activation.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 2009;29(8):1463-74.
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2009: Armstead William M; Cines Douglas B; Bdeir Khalil H; Bdeir Yasmina; Stein Sherman C; Higazi Abd Al-Roof
uPA modulates the age-dependent effect of brain injury on cerebral hemodynamics through LRP and ERK MAPK.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 2009;29(3):524-33.
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2008: Armstead William M; Christine Amy J; Higazi Abd Al-Roof; Cines Douglas B
Urokinase plasminogen activator impairs SNP and PGE2 cerebrovasodilation after brain injury through activation of LRP and ERK MAPK.
Journal of neurotrauma 2008;25(11):1375-81.
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2008: Armstead William M; Cines Douglas B; Bdeir Khalil; Kulikovskaya Irina; Stein Sherman C; Higazi Abd Al-Roof
uPA impairs cerebrovasodilation after hypoxia/ischemia through LRP and ERK MAPK.
Brain research 2008;1231():121-31.
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2007: Armstead William M; Vavilala Monica S
Adrenomedullin reduces gender-dependent loss of hypotensive cerebrovasodilation after newborn brain injury through activation of ATP-dependent K channels.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 2007;27(10):1702-9.
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2006: Armstead William M
Association between intravascular coagulopathy and outcome after traumatic brain injury.
Neurology India 2006;54(4):347-8.
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2006: Armstead William M; Nassar Taher; Akkawi Saed; Smith Douglas H; Chen Xiao-Han; Cines Douglas B; Higazi Abd Al-Roof
Neutralizing the neurotoxic effects of exogenous and endogenous tPA.
Nature neuroscience 2006;9(9):1150-5.
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2006: Armstead William M
Differential activation of ERK, p38, and JNK MAPK by nociceptin/orphanin FQ in the potentiation of prostaglandin cerebrovasoconstriction after brain injury.
European journal of pharmacology 2006;529(1-3):129-35.
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2005: Baranov Dimitry; Armstead William M
Nitric oxide contributes to AT2 but not AT1 angiotensin II receptor-mediated vasodilatation of porcine pial arteries and arterioles.
European journal of pharmacology 2005;525(1-3):112-6.
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2005: Armstead William M; Cines Douglas B; Higazi Abd Al-Roof
Plasminogen activators contribute to impairment of hypercapnic and hypotensive cerebrovasodilation after cerebral hypoxia/ischemia in the newborn pig.
Stroke; a journal of cerebral circulation 2005;36(10):2265-9.
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2005: Armstead William M; Hecker James G
Heat shock protein modulation of KATP and KCa channel cerebrovasodilation after brain injury.
American journal of physiology. Heart and circulatory physiology 2005;289(3):H1184-90.
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2005: Ross John; Armstead William M
NOC/oFQ activates ERK and JNK but not p38 MAPK to impair prostaglandin cerebrovasodilation after brain injury.
Brain research 2005;1054(1):95-102.
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2005: Armstead William M; Cines Douglas B; Higazie Abd Al-Roof
Plasminogen activators contribute to age-dependent impairment of NMDA cerebrovasodilation after brain injury.
Brain research. Developmental brain research 2005;156(2):139-46.
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2004: Armstead William M
NMDA and age dependent cerebral hemodynamics after traumatic brain injury.
Experimental and toxicologic pathology : official journal of the Gesellschaft für Toxikologische Pathologie 2004;56(1-2):75-81.
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2004: Armstead William M; Cines Douglas B; Al-Roof Higazi Abd
Altered NO function contributes to impairment of uPA and tPA cerebrovasodilation after brain injury.
Journal of neurotrauma 2004;21(9):1204-11.
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2004: Philip Shaji; Armstead William M
NMDA dilates pial arteries by KATP and Kca channel activation.
Brain research bulletin 2004;63(2):127-31.
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2004: Ford Jonathan; Armstead William M
Nociceptin/orphanin FQ alters prostaglandin cerebrovascular action following brain injury.
Journal of neurotrauma 2004;21(2):187-93.
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2004: Armstead W M
Endothelins and the role of endothelin antagonists in the management of posttraumatic vasospasm.
Current pharmaceutical design 2004;10(18):2185-92.
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2003: Baranov Dimitry; Armstead William M
Selective blockade of AT1 receptor attenuates impairment of hypotensive autoregulation and improves cerebral blood flow after brain injury in the newborn pig.
Anesthesiology 2003;99(5):1118-24.
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2003: Armstead William M
PTK, ERK and p38 MAPK contribute to impaired NMDA-induced vasodilation after brain injury.
European journal of pharmacology 2003;474(2-3):249-54.
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2003: Philip Shaji; Armstead William M
Differential role of PTK, ERK and p38 MAPK in superoxide impairment of NMDA cerebrovasodilation.
Brain research 2003;979(1-2):98-103.
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2003: Armstead William M
Cyclooxygenase-2-dependent superoxide generation contributes to age-dependent impairment of G protein-mediated cerebrovasodilation.
Anesthesiology 2003;98(6):1378-83.
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2003: Armstead William M
Protein kinase C activation generates superoxide and contributes to impairment of cerebrovasodilation induced by G protein activation after brain injury.
Brain research 2003;971(2):153-60.
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2003: Philip Shaji; Armstead William M
Newborn pig nociceptin/orphanin FQ activates protein tyrosine kinase and mitogen activated protein kinase to impair NMDA cerebrovasodilation after ischemia.
Neuroreport 2003;14(2):201-3.
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2003: Armstead William M
ET-1 contributes to age-dependent G protein impairment after brain injury.
Journal of neurotrauma 2003;20(1):105-10.
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2003: Jagolino Amanda L; Armstead William M
PTK, MAPK, and NOC/oFQ impair hypercapnic cerebrovasodilation after hypoxia/ischemia.
American journal of physiology. Heart and circulatory physiology 2003;284(1):H101-7.
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2002: Armstead William M
Role of Nociceptin/Orphanin FQ in the physiologic and pathologic control of the cerebral circulation.
Experimental biology and medicine (Maywood, N.J.) 2002;227(11):957-68.
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2002: Armstead William M
G protein activation elicits cerebrovasodilation through interaction with K(ATP) and K(Ca) channels.
Brain research 2002;957(2):369-72.
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2002: Baranov Dimitry; Armstead William M
Prostaglandins contribute to impaired angiotensin II-induced cerebral vasodilation after brain injury.
Journal of neurotrauma 2002;19(11):1457-66.
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2002: Armstead William M
Age dependent NMDA contribution to impaired hypotensive cerebral hemodynamics following brain injury.
Brain research. Developmental brain research 2002;139(1):19-28.
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2002: Armstead William M
Age-dependent NOC/oFQ contribution to impaired hypotensive cerebral hemodynamics after brain injury.
Journal of neurotrauma 2002;19(10):1193-202.
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2002: Kulkarni Miriam; Armstead William M
Relationship between NOC/oFQ, dynorphin, and COX-2 activation in impaired NMDA cerebrovasodilation after brain injury.
Journal of neurotrauma 2002;19(8):965-73.
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2002: Armstead William M
Protein tyrosine kinase and mitogen-activated protein kinase activation contribute to K(ATP) and K(ca) channel impairment after brain injury.
Brain research 2002;943(2):276-82.
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2002: Armstead William M
NOC/oFQ and NMDA contribute to piglet hypoxic ischemic hypotensive cerebrovasodilation impairment.
Pediatric research 2002;51(5):586-91.
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2002: Armstead William
NOC/oFQ activates PKC and generates superoxide to impair hypotensive cerebrovasodilation after hypoxia/ischemia.
Medical science monitor : international medical journal of experimental and clinical research 2002;8(1):BR8-BR14.
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2001: Armstead W M
Endothelin-Induced cyclooxygenase-dependent superoxide generation contributes to K+ channel functional impairment after brain injury.
Journal of neurotrauma 2001;18(10):1039-48.
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2001: Armstead W M
Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K(+) channel function impairment after brain injury.
Brain research 2001;910(1-2):19-28.
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2001: Jagolino A; Armstead W M
Nociceptin/orphanin FQ contributes to hypoxic/ischemic impairment of hypercapnic cerebrovasodilation.
Brain research bulletin 2001;55(4):465-8.
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2001: Armstead W M
Age-Dependent vasopressinergic modulation of Noc/oFQ-induced impairment of NMDA cerebrovasodilation after brain injury.
Journal of neurotrauma 2001;18(6):615-23.
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2001: Armstead W M
Vasopressin-induced protein kinase C-dependent superoxide generation contributes to atp-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injury.
Stroke; a journal of cerebral circulation 2001;32(6):1408-14.
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2001: Armstead W M
Age dependent endothelin contribution to NOC/oFQ induced impairment of NMDA cerebrovasodilation after brain injury.
Peptides 2001;22(1):39-46.
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2000: Armstead W M
Role of altered cyclooxygenase metabolism in impaired cerebrovasodilation to nociceptin/orphanin FQ following brain injury.
Brain research bulletin 2000;53(6):807-12.
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2000: Salvucci A; Armstead W M
Vasopressin impairs K(ATP) and K(ca) channel function after brain injury.
Brain research 2000;887(2):406-12.
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2000: Armstead W M
NOC/oFQ PKC-dependent superoxide generation contributes to hypoxic-ischemic impairment of NMDA cerebrovasodilation.
American journal of physiology. Heart and circulatory physiology 2000;279(6):H2678-84.
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2000: Ben-Haim G; Armstead W M
Role of cAMP and K(+) channel-dependent mechanisms in piglet hypoxic/ischemic impaired nociceptin/orphanin FQ-induced cerebrovasodilation.
Brain research 2000;884(1--2):51-8.
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2000: Armstead W M
NOC/oFQ contributes to age-dependent impairment of NMDA-induced cerebrovasodilation after brain injury.
American journal of physiology. Heart and circulatory physiology 2000;279(5):H2188-95.
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2000: Armstead W M
Role of nociceptin/orphanin FQ in age-dependent cerebral hemodynamic effects of brain injury.
Journal of neurotrauma 2000;17(9):751-64.
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2000: Kulkarni M; Armstead W M
Superoxide generation links nociceptin/orphanin FQ (NOC/oFQ) release to impaired N-methyl-D-aspartate cerebrovasodilation after brain injury.
Stroke; a journal of cerebral circulation 2000;31(8):1990-6.
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2000: Armstead W M
Role of NOC/oFQ in impaired opioid-induced pial artery dilation following brain injury.
Brain research 2000;869(1-2):231-5.
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2000: Armstead W M
NOC/oFQ contributes to hypoxic-ischemic impairment of N-methyl-D-aspartate-induced cerebral vasodilation.
Brain research 2000;868(1):48-55.
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2000: Armstead W M
Altered release of prostaglandins contributes to hypoxic/ischemic impairment of NOC/oFQ cerebrovasodilation.
Brain research 2000;859(1):104-12.
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2000: Armstead W M
Relationship between nociceptin/orphanin FQ and cerebral hemodynamics after hypoxia-ischemia in piglets.
American journal of physiology. Heart and circulatory physiology 2000;278(2):H477-83.
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2000: Ben-Haim G; Armstead W M
Stimulus duration-dependent contribution of k(ca) channel activation and cAMP to hypoxic cerebrovasodilation.
Brain research 2000;853(2):330-7.
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1999: Armstead W M
Role of endothelin-1 in age-dependent cerebrovascular hypotensive responses after brain injury.
The American journal of physiology 1999;277(5 Pt 2):H1884-94.
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1999: Armstead W M
Endothelin-1 contributes to normocapnic hyperoxic pial artery vasoconstriction.
Brain research 1999;842(1):252-5.
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1999: Armstead W M
Nociceptin/orphanin FQ dilates pial arteries by K(ATP) and K(ca) channel activation.
Brain research 1999;835(2):315-23.
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1999: Armstead W M
Age-dependent impairment of K(ATP) channel function following brain injury.
Journal of neurotrauma 1999;16(5):391-402.
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1999: Armstead W M
Cerebral hemodynamics after traumatic brain injury of immature brain.
Experimental and toxicologic pathology : official journal of the Gesellschaft für Toxikologische Pathologie 1999;51(2):137-42.
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1999: Armstead W M
Superoxide generation links protein kinase C activation to impaired ATP-sensitive K+ channel function after brain injury.
Stroke; a journal of cerebral circulation 1999;30(1):153-9.
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1998: Venteicher A; Armstead W M
Vasopressin contributes to dynorphin modulation of hypoxic cerebrovasodilation.
The American journal of physiology 1998;275(6 Pt 2):H2072-9.
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1998: Armstead W M
Nitric oxide contributes to opioid release from glia during hypoxia.
Brain research 1998;813(2):398-401.
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1998: Armstead W M
Brain injury impairs prostaglandin cerebrovasodilation.
Journal of neurotrauma 1998;15(9):721-9.
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1998: Armstead W M
Relationship among NO, the KATP channel, and opioids in hypoxic pial artery dilation.
The American journal of physiology 1998;275(3 Pt 2):H988-94.
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1998: Armstead W M
Role of opioids in hypoxic pial artery dilation is stimulus duration dependent.
The American journal of physiology 1998;275(3 Pt 2):H861-7.
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1998: Armstead W M
Contribution of kca channel activation to hypoxic cerebrovasodilation does not involve NO.
Brain research 1998;799(1):44-8.
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1998: Armstead W M
ATP-dependent K+ channel activation reduces loss of opioid dilation after brain injury.
The American journal of physiology 1998;274(5 Pt 2):H1674-83.
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1998: Wilderman M J; Armstead W M
Role of endothelial nitric oxide synthase in hypoxia-induced pial artery dilation.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 1998;18(5):531-8.
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1998: Al-Turki A; Armstead W M
Altered release of prostaglandins by opioids contributes to impaired cerebral hemodynamics following brain injury.
Critical care medicine 1998;26(5):917-25.
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1997: Kasemsri T; Armstead W M
Endothelin impairs ATP-sensitive K+ channel function after brain injury.
The American journal of physiology 1997;273(6 Pt 2):H2639-47.
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1997: Armstead W M
Brain injury impairs ATP-sensitive K+ channel function in piglet cerebral arteries.
Stroke; a journal of cerebral circulation 1997;28(11):2273-9; discussion 2280.
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1997: Wilderman M J; Armstead W M
Role of neuronal NO synthase in relationship between NO and opioids in hypoxia-induced pial artery dilation.
The American journal of physiology 1997;273(4 Pt 2):H1807-15.
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1997: Armstead W M
Role of impaired cAMP and calcium-sensitive K+ channel function in altered cerebral hemodynamics following brain injury.
Brain research 1997;768(1-2):177-84.
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1997: Armstead W M
Role of activation of calcium-sensitive K+ channels in NO- and hypoxia-induced pial artery vasodilation.
The American journal of physiology 1997;272(4 Pt 2):H1785-90.
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1997: Rossberg M I; Armstead W M
Role of cyclic nucleotides in vasopressin-induced piglet pial artery dilation and opioid release.
Pediatric research 1997;41(4 Pt 1):498-504.
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1997: Wilderman M J; Armstead W M
Role of PACAP in the relationship between cAMP and opioids in hypoxia-induced pial artery vasodilation.
The American journal of physiology 1997;272(3 Pt 2):H1350-8.
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1997: Armstead W M
Role of opioids in the physiologic and pathophysiologic control of the cerebral circulation.
Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.) 1997;214(3):210-21.
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1997: Armstead W M
Role of activation of calcium-sensitive K+ channels and cAMP in opioid-induced pial artery dilation.
Brain research 1997;747(2):252-8.
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1997: Kasemsri T; Armstead W M
Endothelin production links superoxide generation to altered opioid-induced pial artery vasodilation after brain injury in pigs.
Stroke; a journal of cerebral circulation 1997;28(1):190-6; discussion 197.
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1997: Armstead W M
Role of nitric oxide, cyclic nucleotides, and the activation of ATP-sensitive K+ channels in the contribution of adenosine to hypoxia-induced pial artery dilation.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 1997;17(1):100-8.
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1996: Armstead W M
Role of endothelin in pial artery vasoconstriction and altered responses to vasopressin after brain injury.
Journal of neurosurgery 1996;85(5):901-7.
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1996: Wilderman M J; Armstead W M
Influence of cAMP on cerebrospinal fluid opioid concentration: role in cAMP-induced pial artery dilation.
European journal of pharmacology 1996;309(3):243-9.
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1996: Rossberg M I; Armstead W M
Relationship between vasopressin and opioids in hypoxia induced pial artery vasodilation.
The American journal of physiology 1996;271(2 Pt 2):H521-7.
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1996: Armstead W M
cGMP and cAMP in prostaglandin-induced pial artery dilation and increased CSF opioid concentration.
The American journal of physiology 1996;271(1 Pt 2):H166-72.
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1996: Armstead W M
Relationship between opioids and prostaglandins in hypoxia-induced vasodilation of pial arteries in the newborn pig.
Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.) 1996;212(2):135-41.
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1996: Armstead W M
Influence of brain injury on vasopressin-induced pial artery vasodilation: role of superoxide anion.
The American journal of physiology 1996;270(4 Pt 2):H1272-8.
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1996: Armstead W M
Role of vasopressin in altered pial artery responses to dynorphin and beta-endorphin following brain injury.
Journal of neurotrauma 1996;13(3):115-23.
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1996: Wilderman M J; Armstead W M
Relationship between nitric oxide and opioids in hypoxia-induced pial artery vasodilation.
The American journal of physiology 1996;270(3 Pt 2):H869-74.
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1996: Thorogood M C; Armstead W M
Influence of polyethylene glycol superoxide dismutase/catalase on altered opioid-induced pial artery dilation after brain injury.
Anesthesiology 1996;84(3):614-25.
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1996: Armstead W M
Role of ATP-sensitive K+ channels in cGMP-mediated pial artery vasodilation.
The American journal of physiology 1996;270(2 Pt 2):H423-6.
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1995: Thorogood M C; Armstead W M
Influence of brain injury on opioid-induced pial artery vasodilation.
The American journal of physiology 1995;269(5 Pt 2):H1776-83.
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1995: Shankar V; Armstead W M
Opioids contribute to hypoxia-induced pial artery dilation through activation of ATP-sensitive K+ channels.
The American journal of physiology 1995;269(3 Pt 2):H997-1002.
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1995: Armstead W M
Relationship between opioids and activation of phospholipase C and protein kinase C in brain injury induced pial artery vasoconstriction.
Brain research 1995;689(2):183-8.
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1995: Armstead W M
The contribution of delta 1- and delta 2-opioid receptors to hypoxia-induced pial artery dilation in the newborn pig.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 1995;15(3):539-46.
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1995: Armstead W M
Role of nitric oxide and cAMP in prostaglandin-induced pial arterial vasodilation.
The American journal of physiology 1995;268(4 Pt 2):H1436-40.
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1995: Devine J O; Armstead W M
The role of nitric oxide in opioid-induced pial artery vasodilation.
Brain research 1995;675(1-2):257-63.
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1995: Rebich S; Devine J O; Armstead W M
Role of nitric oxide and cAMP in beta-adrenoceptor-induced pial artery vasodilation.
The American journal of physiology 1995;268(3 Pt 2):H1071-6.
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1995: Armstead W M
Opioids and nitric oxide contribute to hypoxia-induced pial arterial vasodilation in newborn pigs.
The American journal of physiology 1995;268(1 Pt 2):H226-32.
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1994: Armstead W M; Kurth C D
Different cerebral hemodynamic responses following fluid percussion brain injury in the newborn and juvenile pig.
Journal of neurotrauma 1994;11(5):487-97.
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1994: Armstead W M; Kurth C D
The role of opioids in newborn pig fluid percussion brain injury.
Brain research 1994;660(1):19-26.
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