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Philipp Henneke

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S Farmand; Philipp Henneke; M Hufnagel; R Berner
Significant decline in the erythromycin resistance of group A streptococcus isolates at a German paediatric tertiary care centre.
Markus Hufnagel; Philipp Henneke; Annette Schmitt-Graeff
IgG4-related disease.
Alessandro Costa; Roberta Galbo; Douglas T Golenbock; Rahul Gupta; Philipp Henneke; Antonio Malara; Giuseppe Mancuso; Angelina Midiri; Salvatore Papasergi; Giacomo Signorino; Giuseppe Teti; Patrick Trieu-Cuot; Carmelo Biondo; Francesco Cardile; Concetta Beninati
Activation of the NLRP3 inflammasome by group B streptococci.

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All Publications

2012: S Farmand; Philipp Henneke; M Hufnagel; R Berner
Significant decline in the erythromycin resistance of group A streptococcus isolates at a German paediatric tertiary care centre.
European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology 2012;31(5):707-10.
2012: Markus Hufnagel; Philipp Henneke; Annette Schmitt-Graeff
IgG4-related disease.
The New England journal of medicine 2012;366(17):1643-4; author reply 1646-7.
2012: Alessandro Costa; Roberta Galbo; Douglas T Golenbock; Rahul Gupta; Philipp Henneke; Antonio Malara; Giuseppe Mancuso; Angelina Midiri; Salvatore Papasergi; Giacomo Signorino; Giuseppe Teti; Patrick Trieu-Cuot; Carmelo Biondo; Francesco Cardile; Concetta Beninati
Activation of the NLRP3 inflammasome by group B streptococci.
Journal of immunology (Baltimore, Md. : 1950) 2012;188(4):1953-60.
2012: Susan Farmand; Sybille Kenzel; Elke Lainka; Helga Löffler; Stefan Wirth; Tanja Woltering; Philipp Henneke
Spontaneous clearance of hepatitis C virus in vertically infected children.
European journal of pediatrics 2012;171(2):253-8.
2009: Sandra Santos-Sierra; Sachin D Deshmukh; Julia Kalnitski; Peter Küenzi; Matthias P Wymann; Douglas T Golenbock; Philipp Henneke
Mal connects TLR2 to PI3Kinase activation and phagocyte polarization.
The EMBO journal 2009;28(14):2018-27.
2009: Helga Löffler; Philipp Henneke
[Infection with human immunodeficiency virus (HIV) in childhood].
Kinderkrankenschwester : Organ der Sektion Kinderkrankenpflege / Deutsche Gesellschaft für Sozialpädiatrie und Deutsche Gesellschaft für Kinderheilkunde 2009;28(2):59-69.
2009: Marco Hoffmann; Mirjam B Zeisel; Nikolaus Jilg; Glaucia Paranhos-Baccalà; Françoise Stoll-Keller; Takaji Wakita; Peter Hafkemeyer; Hubert E Blum; Heidi Barth; Philipp Henneke; Thomas F Baumert
Toll-like receptor 2 senses hepatitis C virus core protein but not infectious viral particles.
Journal of innate immunity 2009;1(5):446-54.
2008: Julia Wennekamp; Philipp Henneke
Induction and termination of inflammatory signaling in group B streptococcal sepsis.
Immunological reviews 2008;225():114-27.
2008: Markus Hufnagel; Anita Burger; Susanne Bartelt; Philipp Henneke; Reinhard Berner
Secular trends in pediatric bloodstream infections over a 20-year period at a tertiary care hospital in Germany.
European journal of pediatrics 2008;167(10):1149-59.
2008: Philipp Henneke; Shaynoor Dramsi; Giuseppe Mancuso; Kamila Chraibi; Elisabeth Pellegrini; Christian Theilacker; Johannes Hübner; Sandra Santos-Sierra; Giuseppe Teti; Douglas T Golenbock; Claire Poyart; Patrick Trieu-Cuot
Lipoproteins are critical TLR2 activating toxins in group B streptococcal sepsis.
Journal of immunology (Baltimore, Md. : 1950) 2008;180(9):6149-58.
2007: Seija Lehnardt; Julia Wennekamp; Dorette Freyer; Christian Liedtke; Christina Krueger; Robert Nitsch; Ingo Bechmann; Joerg R Weber; Philipp Henneke
TLR2 and caspase-8 are essential for group B Streptococcus-induced apoptosis in microglia.
Journal of immunology (Baltimore, Md. : 1950) 2007;179(9):6134-43.
2007: Katja M Ubenauf; Marcus Krueger; Philipp Henneke; Reinhard Berner
Lipopolysaccharide binding protein is a potential marker for invasive bacterial infections in children.
The Pediatric infectious disease journal 2007;26(2):159-62.
2006: Kirsten Fluegge; Brit Adams; Urban Luetke Volksbeck; Annerose Serr; Philipp Henneke; Reinhard Berner
Low prevalence of methicillin-resistant Staphylococcus aureus (MRSA) in a southwestern region of Germany.
European journal of pediatrics 2006;165(10):688-90.
2006: Philipp Henneke; Reinhard Berner
SIRS and group-B streptococcal sepsis in newborns: pathogenesis and perspectives in adjunctive therapy.
Seminars in fetal & neonatal medicine 2006;11(5):333-42.
2006: Seija Lehnardt; Philipp Henneke; Egil Lien; Dennis L Kasper; Joseph J Volpe; Ingo Bechmann; Robert Nitsch; Joerg R Weber; Douglas T Golenbock; Timothy Vartanian
A mechanism for neurodegeneration induced by group B streptococci through activation of the TLR2/MyD88 pathway in microglia.
Journal of immunology (Baltimore, Md. : 1950) 2006;177(1):583-92.
2006: Philipp Henneke; Reinhard Berner
Interaction of neonatal phagocytes with group B streptococcus: recognition and response.
Infection and immunity 2006;74(6):3085-95.
2006: Sybille Kenzel; Philipp Henneke
The innate immune system and its relevance to neonatal sepsis.
Current opinion in infectious diseases 2006;19(3):264-70.
2006: Sybille Kenzel; Giuseppe Mancuso; Richard Malley; Giuseppe Teti; Douglas T Golenbock; Philipp Henneke
c-Jun kinase is a critical signaling molecule in a neonatal model of group B streptococcal sepsis.
Journal of immunology (Baltimore, Md. : 1950) 2006;176(5):3181-8.
2006: Sandra Santos-Sierra; Douglas T Golenbock; Philipp Henneke
Toll-like receptor-dependent discrimination of streptococci.
Journal of endotoxin research 2006;12(5):307-12.
2005: Amit Srivastava; Philipp Henneke; Alberto Visintin; Sarah C Morse; Victoria Martin; Claire Watkins; James C Paton; Michael R Wessels; Douglas T Golenbock; Richard Malley
The apoptotic response to pneumolysin is Toll-like receptor 4 dependent and protects against pneumococcal disease.
Infection and immunity 2005;73(10):6479-87.
2005: Philipp Henneke; Siegfried Morath; Satoshi Uematsu; Stefan Weichert; Markus Pfitzenmaier; Osamu Takeuchi; Andrea Müller; Claire Poyart; Shizuo Akira; Reinhard Berner; Giuseppe Teti; Armin Geyer; Thomas Hartung; Patrick Trieu-Cuot; Dennis L Kasper; Douglas T Golenbock
Role of lipoteichoic acid in the phagocyte response to group B streptococcus.
Journal of immunology (Baltimore, Md. : 1950) 2005;174(10):6449-55.
2005: Heidi Barth; Axel Ulsenheimer; Gerd R Pape; Helmut M Diepolder; Marco Hoffmann; Christoph Neumann-Haefelin; Robert Thimme; Philipp Henneke; Reinhild Klein; Glaucia Paranhos-Baccalà; Erik Depla; TSANYANG LIANG; Hubert E Blum; Thomas F Baumert
Uptake and presentation of hepatitis C virus-like particles by human dendritic cells.
Blood 2005;105(9):3605-14.
2004: Anselm Enders; Ulrich Pannicke; Reinhard Berner; Philipp Henneke; Klaus Radlinger; Klaus Schwarz; Stephan Ehl
Two siblings with lethal pneumococcal meningitis in a family with a mutation in Interleukin-1 receptor-associated kinase 4.
The Journal of pediatrics 2004;145(5):698-700.
2004: Øyvind Halaas; Nina Beate Liabakk; R Vik; Concetta Beninati; Philipp Henneke; Anders Sundan; Terje Espevik
Monocytes stimulated with group B streptococci or interferons release tumour necrosis factor-related apoptosis-inducing ligand.
Scandinavian journal of immunology 2004;60(1-2):74-81.
2004: Giuseppe Mancuso; Angelina Midiri; Concetta Beninati; Carmelo Biondo; Roberta Galbo; Shizuo Akira; Philipp Henneke; Douglas T Golenbock; Giuseppe Teti
Dual role of TLR2 and myeloid differentiation factor 88 in a mouse model of invasive group B streptococcal disease.
Journal of immunology (Baltimore, Md. : 1950) 2004;172(10):6324-9.
2003: Keya Sau; Salamatu S Mambula; Eicke Latz; Philipp Henneke; Douglas T Golenbock; Stuart M Levitz
The antifungal drug amphotericin B promotes inflammatory cytokine release by a Toll-like receptor- and CD14-dependent mechanism.
The Journal of biological chemistry 2003;278(39):37561-8.
2003: Richard Malley; Philipp Henneke; Sarah C Morse; Michael J Cieslewicz; Marc Lipsitch; Claudette M Thompson; Evelyn A Kurt-Jones; James C Paton; Michael R Wessels; Douglas T Golenbock
Recognition of pneumolysin by Toll-like receptor 4 confers resistance to pneumococcal infection.
Proceedings of the National Academy of Sciences of the United States of America 2003;100(4):1966-71.
2002: Salamatu S Mambula; Keya Sau; Philipp Henneke; Douglas T Golenbock; Stuart M Levitz
Toll-like receptor (TLR) signaling in response to Aspergillus fumigatus.
The Journal of biological chemistry 2002;277(42):39320-6.
2002: Philipp Henneke; Osamu Takeuchi; Richard Malley; Egil Lien; Robin R Ingalls; Mason W Freeman; Tanya N Mayadas; Victor Nizet; Shizuo Akira; Dennis L Kasper; Douglas T Golenbock
Cellular activation, phagocytosis, and bactericidal activity against group B streptococcus involve parallel myeloid differentiation factor 88-dependent and independent signaling pathways.
Journal of immunology (Baltimore, Md. : 1950) 2002;169(7):3970-7.
2002: Philipp Henneke; Douglas T Golenbock
Innate immune recognition of lipopolysaccharide by endothelial cells.
Critical care medicine 2002;30(5 Suppl):S207-13.
2002: Paola Massari; Philipp Henneke; Yu Ho; Eicke Latz; Douglas T Golenbock; Wetzler Lee
Cutting edge: Immune stimulation by neisserial porins is toll-like receptor 2 and MyD88 dependent.
Journal of immunology (Baltimore, Md. : 1950) 2002;168(4):1533-7.
2001: Philipp Henneke; Osamu Takeuchi; Jos van Strijp; Hilde-Kari Guttormsen; J A Smith; Andra B Schromm; Terje Espevik; Shizuo Akira; V Nizet; Dennis L Kasper; Douglas T Golenbock
Novel engagement of CD14 and multiple toll-like receptors by group B streptococci.
Journal of immunology (Baltimore, Md. : 1950) 2001;167(12):7069-76.
2001: Andrei E Medvedev; Philipp Henneke; Andra B Schromm; Egil Lien; Robin R Ingalls; Matthew J Fenton; Douglas T Golenbock; Stefanie N Vogel
Induction of tolerance to lipopolysaccharide and mycobacterial components in Chinese hamster ovary/CD14 cells is not affected by overexpression of Toll-like receptors 2 or 4.
Journal of immunology (Baltimore, Md. : 1950) 2001;167(4):2257-67.
2001: Andra B Schromm; Egil Lien; Philipp Henneke; J C Chow; A Yoshimura; Holger Heine; Eicke Latz; Brian G Monks; D A Schwartz; K Miyake; Douglas T Golenbock
Molecular genetic analysis of an endotoxin nonresponder mutant cell line: a point mutation in a conserved region of MD-2 abolishes endotoxin-induced signaling.
The Journal of experimental medicine 2001;194(1):79-88.