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Ines Kovacs

Research Profile

Chemicals & Drugs

Disorders

Concepts & Ideas

Anatomy

Procedures

Physiology

Phenomena

False Negative Reactions

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Publications

TOP 3
Stephanie G Willis; Thoralf Lange; Shadmehr Demehri; Sandra Otto; Lucy C Crossman; Dietger Niederwieser; Eric P Stoffregen; Shannon McWeeney; Ines Kovacs; Byung S Park; Brian J Druker; Michael W N Deininger
High-sensitivity detection of BCR-ABL kinase domain mutations in imatinib-naive patients: correlation with clonal cytogenetic evolution but not response to therapy.
Thoralf Lange; Thomas Bumm; Sandra Otto; Haifa-Kathrin Al-Ali; Ines Kovacs; Daniela Krug; Thomas Köhler; Rainer Krahl; Dietger Niederwieser; Michael W N Deininger
Quantitative reverse transcription polymerase chain reaction should not replace conventional cytogenetics for monitoring patients with chronic myeloid leukemia during early phase of imatinib therapy.
Haifa-Kathrin Al-Ali; Sabine Leiblein; Ines Kovacs; Evelin Hennig; Dietger Niederwieser; Michael W N Deininger
CML with an e1a3 BCR-ABL fusion: rare, benign, and a potential diagnostic pitfall.

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All Publications

2005: Stephanie G Willis; Thoralf Lange; Shadmehr Demehri; Sandra Otto; Lucy C Crossman; Dietger Niederwieser; Eric P Stoffregen; Shannon McWeeney; Ines Kovacs; Byung S Park; Brian J Druker; Michael W N Deininger
High-sensitivity detection of BCR-ABL kinase domain mutations in imatinib-naive patients: correlation with clonal cytogenetic evolution but not response to therapy.
Blood 2005;106(6):2128-37.
2004: Thoralf Lange; Thomas Bumm; Sandra Otto; Haifa-Kathrin Al-Ali; Ines Kovacs; Daniela Krug; Thomas Köhler; Rainer Krahl; Dietger Niederwieser; Michael W N Deininger
Quantitative reverse transcription polymerase chain reaction should not replace conventional cytogenetics for monitoring patients with chronic myeloid leukemia during early phase of imatinib therapy.
Haematologica 2004;89(1):49-57.
2002: Haifa-Kathrin Al-Ali; Sabine Leiblein; Ines Kovacs; Evelin Hennig; Dietger Niederwieser; Michael W N Deininger
CML with an e1a3 BCR-ABL fusion: rare, benign, and a potential diagnostic pitfall.
Blood 2002;100(3):1092-3.

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