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Lara Moumné

Research Profile

Chemicals & Drugs

Disorders

Anatomy

Living Beings

Physiology

Concepts & Ideas

Protein Structure, Tertiary

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Publications

TOP 3
Maria G Beconi; Fred Brookfield; Roland W Bürli; Steven Clifton; Lynsey Crowley; Celia Dominguez; Alan F Haughan; Kim Matthews; George McAllister; Lara Moumné; Ignacio Munoz-Sanjuan; Catherine O'Connell; Hannah Pett; Donna L Smith; Julie Vann; Ben Woodman; Dawn Yates; Omar Aziz; Gillian P Bates; Vahri Beaumont
Oral administration of the pimelic diphenylamide HDAC inhibitor HDACi 4b is unsuitable for chronic inhibition of HDAC activity in the CNS in vivo.
Lara Moumné; Yingbin Ouyang; Ken Campbell; David Howland; Gillian P Bates
Genetic knock-down of HDAC3 does not modify disease-related phenotypes in a mouse model of Huntington's disease.
Jeyabalan Nallathambi; Paul Laissue; Frank Batista; Bérénice A Benayoun; Corinne Lesaffre; Lara Moumné; Pj Eswari Pandaranayaka; Kim Usha; Sankaran Krishnaswamy; Periasamy Sundaresan; Reiner A Veitia
Differential functional effects of novel mutations of the transcription factor FOXL2 in BPES patients.

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All Publications

2012: Maria G Beconi; Fred Brookfield; Roland W Bürli; Steven Clifton; Lynsey Crowley; Celia Dominguez; Alan F Haughan; Kim Matthews; George McAllister; Lara Moumné; Ignacio Munoz-Sanjuan; Catherine O'Connell; Hannah Pett; Donna L Smith; Julie Vann; Ben Woodman; Dawn Yates; Omar Aziz; Gillian P Bates; Vahri Beaumont
Oral administration of the pimelic diphenylamide HDAC inhibitor HDACi 4b is unsuitable for chronic inhibition of HDAC activity in the CNS in vivo.
PloS one 2012;7(9):e44498.
2012: Lara Moumné; Yingbin Ouyang; Ken Campbell; David Howland; Gillian P Bates
Genetic knock-down of HDAC3 does not modify disease-related phenotypes in a mouse model of Huntington's disease.
PloS one 2012;7(2):e31080.
2008: Jeyabalan Nallathambi; Paul Laissue; Frank Batista; Bérénice A Benayoun; Corinne Lesaffre; Lara Moumné; Pj Eswari Pandaranayaka; Kim Usha; Sankaran Krishnaswamy; Periasamy Sundaresan; Reiner A Veitia
Differential functional effects of novel mutations of the transcription factor FOXL2 in BPES patients.
Human mutation 2008;29(8):E123-31.
2008: Diane Beysen; Lara Moumné; Reiner A Veitia; Hartmut Peters; Bart P Leroy; Anne De Paepe; Elfride De Baere
Missense mutations in the forkhead domain of FOXL2 lead to subcellular mislocalization, protein aggregation and impaired transactivation.
Human molecular genetics 2008;17(13):2030-8.
2008: Lara Moumné; Aurélie Dipietromaria; Frank Batista; Ayhan Kocer; Marc Fellous; Eric Pailhoux; Reiner A Veitia
Differential aggregation and functional impairment induced by polyalanine expansions in FOXL2, a transcription factor involved in cranio-facial and ovarian development.
Human molecular genetics 2008;17(7):1010-9.
2008: Lara Moumné; Frank Batista; B A Benayoun; Jeyabalan Nallathambi; Marc Fellous; Sundaresan Periasamy; Reiner A Veitia
The mutations and potential targets of the forkhead transcription factor FOXL2.
Molecular and cellular endocrinology 2008;282(1-2):2-11.
2007: Jeyabalan Nallathambi; Lara Moumné; Elfride De Baere; Diane Beysen; Kim Usha; Sundaresan Periasamy; Reiner A Veitia
A novel polyalanine expansion in FOXL2: the first evidence for a recessive form of the blepharophimosis syndrome (BPES) associated with ovarian dysfunction.
Human genetics 2007;121(1):107-12.
2006: Lara Moumné; Reiner A Veitia
[Translational restart downstream of a premature stop codon and protein aggregation].
Médecine sciences : M/S 2006;22(3):232-4.
2005: Lara Moumné; Marc Fellous; Reiner A Veitia
Deletions in the polyAlanine-containing transcription factor FOXL2 lead to intranuclear aggregation.
Human molecular genetics 2005;14(23):3557-64.
2004: Sandrine Caburet; A Demarez; Lara Moumné; Marc Fellous; Elfride De Baere; Reiner A Veitia
A recurrent polyalanine expansion in the transcription factor FOXL2 induces extensive nuclear and cytoplasmic protein aggregation.
Journal of medical genetics 2004;41(12):932-6.